NCT02696304

Brief Summary

Along with the improvement of childhood acute leukemia treatment, survival rates have increased. Therefore, the number of long term childhood leukemia survivors has increased progressively over the last decades. So, the assessment of long term health status in this population becomes very important. Many studies have shown an increased risk of life threatening late complications and early mortality. Cardiovascular morbidity and mortality are particularly frequent. Among these late complications, the metabolic syndrome (MS) is an important concern since it is associated with cardiovascular morbidity and mortality. The overall MS prevalence in the French prospective cohort of survivors of childhood acute leukemia was 9.2% and 18.6% in cases of total body irradiation (TBI) during the leukemia treatment. Since the median age at MS evaluation was 21 years, this prevalence was very high. Anyway, the MS pathophysiology in this population is still poorly understood. One of the most recent hypothesis about the MS mechanism is based on the adipose tissue inability to store fatty acids: when adipose tissue cannot expanse further to store excess nutriments then lipids accumulate in other tissues. This ectopic lipids accumulation can cause insulin resistance and MS. The investigators hypothesized that the adipose tissue could be damaged by treatments received during childhood acute leukemia treatment (particularly TBI). This leads to morphological and functional abnormalities that could promote the insulin resistance and MS. This ectopic adipose tissue contains less preadipocytes, which could impair its functional properties. The primary endpoint of this study is to compare the morphological and functional characteristics of adipose tissue in patients with a MS who received or not TBI during childhood leukemia treatment . This comparison will focus on:

  • The adipose tissue repartition and evaluation of the ectopic adipose tissue
  • Fibrosis and inflammation of the adipose tissue
  • Preadipocytes quantification The secondary endpoint is to describe:
  • for the whole cohort of included patients,
  • the clinical and biological characteristics associated with the MS.
  • Cardiovascular risk factors and nutritional statement
  • Anthropometric measurements
  • Detection of other endocrinal abnormalities possibly associated with the MS
  • Analysis of inflammation blood markers and adipokines quantification.

Trial Health

43
At Risk

Trial Health Score

Automated assessment based on enrollment pace, timeline, and geographic reach

Trial has exceeded expected completion date
Enrollment
30

participants targeted

Target at below P25 for not_applicable

Timeline
Completed

Started May 2015

Geographic Reach
1 country

1 active site

Status
unknown

Health score is calculated from publicly available data and should be used for screening purposes only.

Trial Relationships

Click on a node to explore related trials.

Study Timeline

Key milestones and dates

Study Start

First participant enrolled

May 1, 2015

Completed
6 months until next milestone

First Submitted

Initial submission to the registry

October 16, 2015

Completed
5 months until next milestone

First Posted

Study publicly available on registry

March 2, 2016

Completed
2 months until next milestone

Primary Completion

Last participant's last visit for primary outcome

May 1, 2016

Completed
7 months until next milestone

Study Completion

Last participant's last visit for all outcomes

December 1, 2016

Completed
Last Updated

March 2, 2016

Status Verified

December 1, 2015

Enrollment Period

1 year

First QC Date

October 16, 2015

Last Update Submit

March 1, 2016

Conditions

Outcome Measures

Primary Outcomes (4)

  • Percent of fat mass (Biphotonic absorptiometry)

    1 year

  • Evaluation of the amount of intra liver and pancreatic triglycerides (%) (proton spectroscopy, expressed as percent related to liver or pancreatic water content)

    1 year

  • Fibrosis and inflammation analyses of the adipose tissu : fibrosis and inflammation gene expression analyses by RQ-PCR

    1 year

  • Preadipocytes quantification in the adipose tissue by immunohistochemistery, expressed as percent of stroma vascular fraction of the adipose tissue

    1 year

Study Arms (2)

TBI for childhood leukemia

OTHER

Patients with a metabolic syndrom who received TBI.

Other: Adipose tissu repartitionOther: Visceral and liver adipose tissue repartitionOther: Preadipocyte quantification and adipose tissue inflamationOther: Inflamation blood markers quantification

No TBI for childhood leukemia

OTHER

Patients with a metabolic syndrom without previousTBI

Other: Adipose tissu repartitionOther: Visceral and liver adipose tissue repartitionOther: Preadipocyte quantification and adipose tissue inflamationOther: Inflamation blood markers quantification

Interventions

absorptiometry,

No TBI for childhood leukemiaTBI for childhood leukemia

MRI,spectroscopy,

No TBI for childhood leukemiaTBI for childhood leukemia

biopsy

No TBI for childhood leukemiaTBI for childhood leukemia

blood drawn,

No TBI for childhood leukemiaTBI for childhood leukemia

Eligibility Criteria

Age18 Years+
Sexall
Healthy VolunteersNo
Age GroupsAdult (18-64), Older Adult (65+)

You may qualify if:

  • Age superior or equal to 18 years
  • Metabolic syndrome: at least 3 criteria among the following:
  • Waist circumference ≥ 102 cm for male and ≥ 88 cm for female)
  • High triglyceride level ≥ 150 mg/dl (1,7 mmol/l) or undergoing treatment for that affection
  • Low HDL-Cholesterol \< 40 mg/dl (1,03 mmol/l) for male ; \< 50 mg/dl (1,3 mmol/l) for femal, or undergoing treatment for that affection
  • Elevated blood pressure: systolic ≥ 130 mmHg and/or diastoloic ≥ 85 mmHg or undergoing treatment for that affection
  • Elevated fasten glucose≥ 100 mg/dl or undergoing treatment for that affection
  • Acute leukemia during childhood (under 18 years of age at the time of leukemia diagnosis)
  • Informed consent obtained

You may not qualify if:

  • pregnancy
  • incomplete evaluation of metabolic syndrome

Contact the study team to confirm eligibility.

Sponsors & Collaborators

Study Sites (1)

Assistance Publique Hopitaux de Marseille

Marseille, 13354, France

RECRUITING

MeSH Terms

Conditions

Metabolic Syndrome

Condition Hierarchy (Ancestors)

Insulin ResistanceHyperinsulinismGlucose Metabolism DisordersMetabolic DiseasesNutritional and Metabolic Diseases

Study Officials

  • Urielle DESALBRES

    AP-HM

    STUDY DIRECTOR
  • Claire OUDIN, MD

    AP-HM

    PRINCIPAL INVESTIGATOR

Central Study Contacts

Claire OUDIN, MD

CONTACT

Study Design

Study Type
interventional
Phase
not applicable
Allocation
NON RANDOMIZED
Masking
NONE
Purpose
DIAGNOSTIC
Intervention Model
PARALLEL
Sponsor Type
OTHER
Responsible Party
SPONSOR

Study Record Dates

First Submitted

October 16, 2015

First Posted

March 2, 2016

Study Start

May 1, 2015

Primary Completion

May 1, 2016

Study Completion

December 1, 2016

Last Updated

March 2, 2016

Record last verified: 2015-12

Locations