NCT02665117

Brief Summary

Chlorthalidone (CTD) may produce various metabolic disturbances, including hypokalemia, activation of Renin-Angiotensin- Aldosterone (RAA) system, oxidative stress, dyslipidemia, Fibroblast growth factor 23 (FGF23) synthesis, and magnesium depletion. These factors may interact with each other to contribute to the development of insulin resistances and metabolic syndrome. Smaller studies have suggested that Potassium magnesium Citrate (KMgCit) can ameliorate CTD- induced metabolic side effects independent of correction of hypokalemia. This study will tests if KMgCit ameliorates CTD induced metabolic effects independent of correction of hypokalemia.

Trial Health

87
On Track

Trial Health Score

Automated assessment based on enrollment pace, timeline, and geographic reach

Enrollment
61

participants targeted

Target at P25-P50 for not_applicable hypertension

Timeline
Completed

Started Jan 2015

Longer than P75 for not_applicable hypertension

Geographic Reach
1 country

3 active sites

Status
completed

Health score is calculated from publicly available data and should be used for screening purposes only.

Trial Relationships

Click on a node to explore related trials.

Study Timeline

Key milestones and dates

Study Start

First participant enrolled

January 1, 2015

Completed
1.1 years until next milestone

First Submitted

Initial submission to the registry

January 22, 2016

Completed
5 days until next milestone

First Posted

Study publicly available on registry

January 27, 2016

Completed
6.8 years until next milestone

Primary Completion

Last participant's last visit for primary outcome

November 4, 2022

Completed
Same day until next milestone

Study Completion

Last participant's last visit for all outcomes

November 4, 2022

Completed
12 months until next milestone

Results Posted

Study results publicly available

October 25, 2023

Completed
Last Updated

October 25, 2023

Status Verified

October 1, 2023

Enrollment Period

7.8 years

First QC Date

January 22, 2016

Results QC Date

September 26, 2023

Last Update Submit

October 23, 2023

Conditions

Keywords

HypokalemiaIsoprostanesInsulin resistanceAldosteroneHepatic fatMuscle MagnesiumLiver fat

Outcome Measures

Primary Outcomes (1)

  • Change in Fasting Plasma Glucose From Week 4 to Week 16

    Fasting plasma glucose was measured from venous blood sample at week 4 and week 16

    week 4 and week 16

Secondary Outcomes (3)

  • Chang in Hepatic Fat Measured at Baseline and Week 16

    baseline to week 16

  • Change in Muscle Magnesium Content Measured at Baseline and Week 16

    baseline to week 16

  • Change in FGF23 From Week 4 to Week 16

    week 4 to week 16

Study Arms (2)

KMgCit + Chlorthalidone

ACTIVE COMPARATOR

After a run-in period of 2-3 weeks on Chlorthalidone, patients will be randomized to the addition of KMgCit for 4 months.

Drug: Potassium Magnesium Citrate (KMgCit)Drug: Chlorthalidone

KCl + Chlorthalidone

ACTIVE COMPARATOR

After a run-in period of 2-3 weeks on Chlorthalidone, patients will be randomized to the addition of KCl for 4 months.

Drug: Potassium Chloride (KCl)Drug: Chlorthalidone

Interventions

KMgCit will be administer for 4 months with chlorthalidone.

Also known as: KMgCit
KMgCit + Chlorthalidone

KCl will be administer for 4 months with chlorthalidone.

Also known as: KCl
KCl + Chlorthalidone

Chlorthalidone will be administered for 2-3 weeks. Then either KCL or KMgCit will be added to Chlorthalidone and the combination will be taken for 4 months.

KCl + ChlorthalidoneKMgCit + Chlorthalidone

Eligibility Criteria

Age21 Years+
Sexall
Healthy VolunteersNo
Age GroupsAdult (18-64), Older Adult (65+)

You may qualify if:

  • Treated or untreated stage I hypertension

You may not qualify if:

  • Diabetes mellitus,
  • Renal impairment (serum creatinine \> 1.4 mg/dL),
  • Any heart diseases such as congestive heart failure, sustained arrhythmia, or coronary heart disease,
  • Chronic regular NSAID use,
  • Allergy to thiazide diuretics,
  • Gastro-esophageal reflux disease (GERD) requiring treatment with acid reducing agents or antacid more than once a week,
  • Esophageal-gastric ulcer or history of gastrointestinal bleeding,
  • Chronic diarrhea, vomiting,
  • Excessive sweating,
  • Unprovoked hypokalemia (serum K \< 3.5 mmol/L) or hyperkalemia (serum K \> 5.3 mmol/L),
  • Abnormal liver function test (Aspartate transaminase (AST) or Alanine transaminase (ALT) above upper limit of normal range),
  • Subjects on any potassium supplement on a regular basis for any reason, such as patients with primary aldosteronism,
  • Pregnancy,
  • History of major depression, bipolar disorder, or schizophrenia,
  • History of substance abuse,
  • +5 more criteria

Contact the study team to confirm eligibility.

Sponsors & Collaborators

Study Sites (3)

University of Texas Southwestern Medical Center at Dallas

Dallas, Texas, 75390, United States

Location

University of Texas Southwestern Medical Center

Dallas, Texas, 75390, United States

Location

UT Southwestern Medical Center

Dallas, Texas, 75390, United States

Location

Related Publications (10)

  • Ahmed LA. Protective effects of magnesium supplementation on metabolic energy derangements in lipopolysaccharide-induced cardiotoxicity in mice. Eur J Pharmacol. 2012 Nov 5;694(1-3):75-81. doi: 10.1016/j.ejphar.2012.07.036. Epub 2012 Aug 23.

    PMID: 22939974BACKGROUND
  • Carter BL, Einhorn PT, Brands M, He J, Cutler JA, Whelton PK, Bakris GL, Brancati FL, Cushman WC, Oparil S, Wright JT Jr; Working Group from the National Heart, Lung, and Blood Institute. Thiazide-induced dysglycemia: call for research from a working group from the national heart, lung, and blood institute. Hypertension. 2008 Jul;52(1):30-6. doi: 10.1161/HYPERTENSIONAHA.108.114389. Epub 2008 May 26. No abstract available.

    PMID: 18504319BACKGROUND
  • Guerrero-Romero F, Rodriguez-Moran M. Hypomagnesemia, oxidative stress, inflammation, and metabolic syndrome. Diabetes Metab Res Rev. 2006 Nov-Dec;22(6):471-6. doi: 10.1002/dmrr.644.

    PMID: 16598698BACKGROUND
  • Hata A, Doi Y, Ninomiya T, Mukai N, Hirakawa Y, Hata J, Ozawa M, Uchida K, Shirota T, Kitazono T, Kiyohara Y. Magnesium intake decreases Type 2 diabetes risk through the improvement of insulin resistance and inflammation: the Hisayama Study. Diabet Med. 2013 Dec;30(12):1487-94. doi: 10.1111/dme.12250. Epub 2013 Jun 29.

    PMID: 23758216BACKGROUND
  • Koenig K, Padalino P, Alexandrides G, Pak CY. Bioavailability of potassium and magnesium, and citraturic response from potassium-magnesium citrate. J Urol. 1991 Feb;145(2):330-4. doi: 10.1016/s0022-5347(17)38330-1.

    PMID: 1988724BACKGROUND
  • Odvina CV, Mason RP, Pak CY. Prevention of thiazide-induced hypokalemia without magnesium depletion by potassium-magnesium-citrate. Am J Ther. 2006 Mar-Apr;13(2):101-8. doi: 10.1097/01.mjt.0000149922.16098.c0.

    PMID: 16645424BACKGROUND
  • Pak CY. Correction of thiazide-induced hypomagnesemia by potassium-magnesium citrate from review of prior trials. Clin Nephrol. 2000 Oct;54(4):271-5.

    PMID: 11076102BACKGROUND
  • Price AL, Lingvay I, Szczepaniak EW, Wiebel J, Victor RG, Szczepaniak LS. The metabolic cost of lowering blood pressure with hydrochlorothiazide. Diabetol Metab Syndr. 2013 Jul 9;5(1):35. doi: 10.1186/1758-5996-5-35.

    PMID: 23837919BACKGROUND
  • Ruml LA, Pak CY. Effect of potassium magnesium citrate on thiazide-induced hypokalemia and magnesium loss. Am J Kidney Dis. 1999 Jul;34(1):107-13. doi: 10.1016/s0272-6386(99)70115-0.

    PMID: 10401023BACKGROUND
  • Vongpatanasin W, Giacona JM, Pittman D, Murillo A, Khan G, Wang J, Johnson T, Ren J, Moe OW, Pak CCY. Potassium Magnesium Citrate Is Superior to Potassium Chloride in Reversing Metabolic Side Effects of Chlorthalidone. Hypertension. 2023 Dec;80(12):2611-2620. doi: 10.1161/HYPERTENSIONAHA.123.21932. Epub 2023 Oct 17.

MeSH Terms

Conditions

HypertensionHypokalemiaInsulin ResistanceFatty Liver

Interventions

potassium-magnesium citratePotassium, DietaryChlorthalidone

Condition Hierarchy (Ancestors)

Vascular DiseasesCardiovascular DiseasesWater-Electrolyte ImbalanceMetabolic DiseasesNutritional and Metabolic DiseasesHyperinsulinismGlucose Metabolism DisordersLiver DiseasesDigestive System Diseases

Intervention Hierarchy (Ancestors)

Potassium CompoundsInorganic ChemicalsBenzenesulfonamidesSulfonamidesAmidesOrganic ChemicalsBenzene DerivativesHydrocarbons, AromaticHydrocarbons, CyclicHydrocarbonsBenzophenonesPhthalimidesImidesKetonesSulfonesSulfur CompoundsIsoindolesHeterocyclic Compounds, 2-RingHeterocyclic Compounds, Fused-RingHeterocyclic Compounds

Results Point of Contact

Title
Dr. Wanpen Vongpatanasin
Organization
UT Southwestern Medical Center

Study Officials

  • Wanpen Vongpatanasin, MD

    University of Texas Southwestern Medical Center

    PRINCIPAL INVESTIGATOR

Publication Agreements

PI is Sponsor Employee
Yes

Study Design

Study Type
interventional
Phase
not applicable
Allocation
RANDOMIZED
Masking
DOUBLE
Who Masked
PARTICIPANT, INVESTIGATOR
Purpose
PREVENTION
Intervention Model
PARALLEL
Sponsor Type
OTHER
Responsible Party
PRINCIPAL INVESTIGATOR
PI Title
Professor

Study Record Dates

First Submitted

January 22, 2016

First Posted

January 27, 2016

Study Start

January 1, 2015

Primary Completion

November 4, 2022

Study Completion

November 4, 2022

Last Updated

October 25, 2023

Results First Posted

October 25, 2023

Record last verified: 2023-10

Locations