NCT02324452

Brief Summary

In this study it will be determined whether the rate of severe toxicity associated with fluoropyrimidine treatment (capecitabine or 5-fluorouracil) can be significantly diminished by individualized dosing of fluoropyrimidines based on upfront genotypic assessment of dihydropyrimidine dehydrogenase (DPD) deficiency. In addition to the genotyping, the DPD phenotype of all patients will be determined by measuring the baseline dihydrouracil/uracil (DHU/U) ratio, in order to investigate whether phenotype-guided treatment can further improve patient safety. In a subgroup of patients, other phenotyping methods will be tested: measuring the plasma levels of uracil after a uracil test dose and a uracil breath test after a dose of \[2-13C\] -labeled uracil. To validate these tests, these phenotyping results will be compared with the results of a DPD activity assay (which measures DPD enzyme activity in peripheral blood mononuclear cells), which is considered the gold standard in measuring DPD phenotype.

Trial Health

87
On Track

Trial Health Score

Automated assessment based on enrollment pace, timeline, and geographic reach

Enrollment
1,103

participants targeted

Target at P75+ for not_applicable

Timeline
Completed

Started Mar 2015

Typical duration for not_applicable

Geographic Reach
1 country

17 active sites

Status
completed

Health score is calculated from publicly available data and should be used for screening purposes only.

Trial Relationships

Click on a node to explore related trials.

Study Timeline

Key milestones and dates

First Submitted

Initial submission to the registry

December 9, 2014

Completed
15 days until next milestone

First Posted

Study publicly available on registry

December 24, 2014

Completed
2 months until next milestone

Study Start

First participant enrolled

March 1, 2015

Completed
2.8 years until next milestone

Primary Completion

Last participant's last visit for primary outcome

January 1, 2018

Completed
2 months until next milestone

Study Completion

Last participant's last visit for all outcomes

March 1, 2018

Completed
Last Updated

May 11, 2018

Status Verified

May 1, 2018

Enrollment Period

2.8 years

First QC Date

December 9, 2014

Last Update Submit

May 7, 2018

Conditions

Outcome Measures

Primary Outcomes (1)

  • Safety: incidence of severe treatment-related toxicity (CTC grade 3 to 5)

    The incidence of severe treatment-related toxicity (CTC grade 3 to 5) in patients carrying DPYD variants compared to wild type patients and compared to a historical cohort of DPYD heterozygous patients treated with a full dose of fluoropyrimidines

    patients will be followed during fluoropyrimidine treatment, expected average of 1 year

Secondary Outcomes (3)

  • Cost-effectiveness: medical costs that are made during fluoropyrimidine treatment seen from a health care perspective

    patients will be followed during fluoropyrimidine treatment, expected average of 1 year

  • DPD phenotype, defined as deficient or not deficient

    Prior to start of fluoropyrimidine treatment of the patient (pre dose)

  • Assessment of pharmacokinetics: Such profile parameters will include Cmax, Tmax, AUC and elimination half-life

    At first week of start of fluoropyrimidine treatment of the patient

Study Arms (2)

heterozygous carrier of DPYD variant

EXPERIMENTAL

Patients screened for four single nucleotide polymorphisms (SNPs) in DPYD (DPYD\*2A, c.2846A\>T, c.1236G\>A/HapB3 and DPYD\*13) that are found to be heterozygous for one of these SNPs

Drug: Fluoropyrimidine (capecitabine or 5-fluorouracil)

wild type for DPYD

EXPERIMENTAL

Patients screened for four single nucleotide polymorphisms (SNPs) in DPYD (DPYD\*2A, c.2846A\>T, c.1236G\>A/HapB3 and DPYD\*13) that are found to be wild type for these SNPs

Drug: Fluoropyrimidine (capecitabine or 5-fluorouracil)

Interventions

Patient that are a heterozygous carrier of a DPYD variant will receive a reduced dosage of capecitabine or 5-fluorouracil (25-50% reduction, depending on which SNP is identified). The dose will be titrated in subsequent cycles, to achieve maximal safe exposure. Patients that are wild type (not carrying any of the for DPYD variants) will receive a normal (full) dose.

Also known as: Xeloda, Capecitabine, 5-fluorouracil, fluorouracil, 5-FU
heterozygous carrier of DPYD variantwild type for DPYD

Eligibility Criteria

Age18 Years+
Sexall
Healthy VolunteersNo
Age GroupsAdult (18-64), Older Adult (65+)

You may qualify if:

  • Pathologically confirmed malignancy for which treatment with a fluoropyrimidine is considered to be in the patient's best interest
  • Age ≥ 18 years
  • Able and willing to give written informed consent
  • WHO performance status of 0, 1 or 2
  • Life expectancy of at least 12 weeks
  • Able to swallow and retain oral medication
  • Able and willing to undergo blood sampling for pharmacogenetic and phenotyping analysis
  • Minimal acceptable safety laboratory values (ANC, platelet count, hepatic function, renal function)
  • Able and willing to undergo blood sampling and breath sampling at several time points
  • Able and willing to receive uracil for the test dose assay
  • Able and willing to receive \[2-13C\] -labeled uracil for the breath test

You may not qualify if:

  • Prior treatment with fluoropyrimidines
  • Patients with known substance abuse, psychotic disorders, and/or other diseases expected to interfere with study or the patient's safety
  • Women who are pregnant or breast feeding
  • Both men and women who refuse to use reliable contraceptive methods throughout the study (adequate contraceptive methods are: condom, sterilization, other barrier contraceptive measures preferably in combination with condoms)
  • Patients with a homozygous polymorphic genotype or compound heterozygous genotype for DPYD

Contact the study team to confirm eligibility.

Sponsors & Collaborators

Study Sites (17)

Netherlands Cancer Institute - Antoni van Leeuwenhoek Hospital

Amsterdam, Netherlands

Location

Wilhelmina Hospital Assen

Assen, Netherlands

Location

Amphia Hospital

Breda, Netherlands

Location

Reinier de Graaf Hospital

Delft, Netherlands

Location

Deventer Hospital

Deventer, Netherlands

Location

Hospital Gelderse Vallei

Ede, Netherlands

Location

Catharina Hospital

Eindhoven, Netherlands

Location

Leiden University Medical Center

Leiden, Netherlands

Location

Maastricht University Medical Center

Maastricht, Netherlands

Location

Canisius-Wilhelmina Hospital

Nijmegen, Netherlands

Location

Laurentius Hospital

Roermond, Netherlands

Location

Bravis Hospital

Roosendaal, Netherlands

Location

Erasmus MC

Rotterdam, Netherlands

Location

Franciscus Gasthuis & Vlietland

Rotterdam, Netherlands

Location

Haga Hospital

The Hague, Netherlands

Location

Medical Center Haaglanden

The Hague, Netherlands

Location

University Medical Center Utrecht

Utrecht, Netherlands

Location

Related Publications (4)

  • Knikman JE, Lopez-Yurda M, Meulendijks D, Deenen MJ, Schellens JHM, Beijnen J, Cats A, Guchelaar HJ. A Nomogram to Predict Severe Toxicity in DPYD Wild-Type Patients Treated With Capecitabine-Based Anticancer Regimens. Clin Pharmacol Ther. 2024 Feb;115(2):269-277. doi: 10.1002/cpt.3100. Epub 2023 Nov 29.

  • Knikman JE, Wilting TA, Lopez-Yurda M, Henricks LM, Lunenburg CATC, de Man FM, Meulendijks D, Nieboer P, Droogendijk HJ, Creemers GJ, Mandigers CMPW, Imholz ALT, Mathijssen RHJ, Portielje JEA, Valkenburg-van Iersel L, Vulink A, van der Poel MHW, Baars A, Swen JJ, Gelderblom H, Schellens JHM, Beijnen JH, Guchelaar HJ, Cats A. Survival of Patients With Cancer With DPYD Variant Alleles and Dose-Individualized Fluoropyrimidine Therapy-A Matched-Pair Analysis. J Clin Oncol. 2023 Dec 10;41(35):5411-5421. doi: 10.1200/JCO.22.02780. Epub 2023 Aug 28.

  • Henricks LM, Lunenburg CATC, de Man FM, Meulendijks D, Frederix GWJ, Kienhuis E, Creemers GJ, Baars A, Dezentje VO, Imholz ALT, Jeurissen FJF, Portielje JEA, Jansen RLH, Hamberg P, Ten Tije AJ, Droogendijk HJ, Koopman M, Nieboer P, van de Poel MHW, Mandigers CMPW, Rosing H, Beijnen JH, Werkhoven EV, van Kuilenburg ABP, van Schaik RHN, Mathijssen RHJ, Swen JJ, Gelderblom H, Cats A, Guchelaar HJ, Schellens JHM. DPYD genotype-guided dose individualisation of fluoropyrimidine therapy in patients with cancer: a prospective safety analysis. Lancet Oncol. 2018 Nov;19(11):1459-1467. doi: 10.1016/S1470-2045(18)30686-7. Epub 2018 Oct 19.

  • Jacobs BA, Deenen MJ, Pluim D, van Hasselt JG, Krahenbuhl MD, van Geel RM, de Vries N, Rosing H, Meulendijks D, Burylo AM, Cats A, Beijnen JH, Huitema AD, Schellens JH. Pronounced between-subject and circadian variability in thymidylate synthase and dihydropyrimidine dehydrogenase enzyme activity in human volunteers. Br J Clin Pharmacol. 2016 Sep;82(3):706-16. doi: 10.1111/bcp.13007. Epub 2016 Jun 3.

MeSH Terms

Conditions

Neoplasms

Interventions

CapecitabineFluorouracil

Intervention Hierarchy (Ancestors)

DeoxycytidineCytidinePyrimidine NucleosidesPyrimidinesHeterocyclic Compounds, 1-RingHeterocyclic CompoundsUracilPyrimidinonesDeoxyribonucleosidesNucleosidesNucleic Acids, Nucleotides, and Nucleosides

Study Officials

  • JHM Schellens, MD, PhD

    The Netherlands Cancer Institute

    PRINCIPAL INVESTIGATOR

Study Design

Study Type
interventional
Phase
not applicable
Allocation
NON RANDOMIZED
Masking
NONE
Purpose
TREATMENT
Intervention Model
SINGLE GROUP
Sponsor Type
OTHER
Responsible Party
SPONSOR

Study Record Dates

First Submitted

December 9, 2014

First Posted

December 24, 2014

Study Start

March 1, 2015

Primary Completion

January 1, 2018

Study Completion

March 1, 2018

Last Updated

May 11, 2018

Record last verified: 2018-05

Locations