NCT05941975

Brief Summary

Multiple sclerosis (MS) is a chronic inflammatory and degenerative disease of the central nervous system (CNS), characterized by a complex interplay of inflammatory demyelination and neuronal damage. The core MS phenotypes defined by clinical course are the relapsing and the progressive forms.Relapsing MS (RMS) is characterized by attacks - also called relapses - defined as new or increasing neurologic dysfunction, followed by periods of partial or complete recovery, without apparent progression of the disease during the periods of remission. In contrast, progressive MS (PMS) is characterized by progressive worsening of neurologic function leading to accumulation of disability over time independent of relapses. Additional descriptors ("active/not-active") serve to better characterize the presence of clinical and/or radiological activity both in relapsing and progressive forms. In recent years, the concept of a silent progression, also known as smouldering MS, is making its way into the common lexicon of MS experts, challenging the current definitions of MS phenotypes. A growing body of literature suggests that the line between RMS and PMS is not as marked as men thought, and that inflammation and neurodegeneration can represent a single disease continuum coexisting early on in the disease course. Whilst it is established that relapse-associated worsening (RAW) can be accounted for by an acute inflammatory focal damage leading to axonal transection and conduction block, the physiopathology underlying the progression independent of relapse activity (PIRA) remains unclear. It is becoming apparent that there is an increasing need for a personalized therapeutic approach by considering the individual MS phenotype of each patient, thereby enabling the choice of the molecule best suited to counteract the predominant disease pattern of that individual patient. There is a limited number of studies combining clinical scores, neurophysiological evaluation and neuroimaging in patients with MS experiencing PIRA. Integrating a multimodal exploration of these patients might allow a step forward in the early recognition, management, and treatment of disability accumulation independent from relapses in patients with MS.

Trial Health

43
At Risk

Trial Health Score

Automated assessment based on enrollment pace, timeline, and geographic reach

Trial has exceeded expected completion date
Enrollment
60

participants targeted

Target at P50-P75 for not_applicable multiple-sclerosis

Timeline
Completed

Started Feb 2023

Typical duration for not_applicable multiple-sclerosis

Geographic Reach
1 country

1 active site

Status
unknown

Health score is calculated from publicly available data and should be used for screening purposes only.

Trial Relationships

Click on a node to explore related trials.

Study Timeline

Key milestones and dates

Study Start

First participant enrolled

February 14, 2023

Completed
3 months until next milestone

First Submitted

Initial submission to the registry

May 15, 2023

Completed
2 months until next milestone

First Posted

Study publicly available on registry

July 12, 2023

Completed
2.5 years until next milestone

Primary Completion

Last participant's last visit for primary outcome

December 31, 2025

Completed
Same day until next milestone

Study Completion

Last participant's last visit for all outcomes

December 31, 2025

Completed
Last Updated

July 12, 2023

Status Verified

July 1, 2023

Enrollment Period

2.9 years

First QC Date

May 15, 2023

Last Update Submit

July 4, 2023

Conditions

Outcome Measures

Primary Outcomes (10)

  • Visual Evoked Potential (VEP)

    To assess the integrity of visual pathways through the optic nerves to the visual cortex.

    Change from baseline to 12 months

  • Somatosensory evoked potential (SSEP)

    To assess the integrity of sensitive pathways through the peripheral nerves and dorsal spinal cord to the somatosensory cortex.

    Change from baseline to 12 months

  • Transcranial magnetic motor evoked potentials (TCmMEP)

    To measure the integrity of motor pathways.

    Change from baseline to 12 months

  • Tesla Brain MRI

    Tesla Brain MRI (descriptive outcome)

    Baseline

  • Neurofilament light chain (NfL) serum levels

    Neurofilament light chain (NfL) serum levels

    Baseline

  • Neurofilament light chain (NfL) serum levels

    Neurofilament light chain (NfL) serum levels

    6 months after baseline

  • Neurofilament light chain (NfL) serum levels

    Neurofilament light chain (NfL) serum levels

    12 months after baseline

  • Epstein-Barr virus (EBV) serology (VCA IgG)

    EBV serology will be assessed (VCA IgG) in the serum of patients to evaluate the variation of antibody titers over time (at baseline, at 6- and 12-month follow-up), and compare to titers at the time of diagnosis (when available in their medical record).

    Baseline

  • Epstein-Barr virus (EBV) serology (VCA IgG)

    EBV serology will be assessed (VCA IgG) in the serum of patients to evaluate the variation of antibody titers over time (at baseline, at 6- and 12-month follow-up), and compare to titers at the time of diagnosis (when available in their medical record).

    6 months after baseline

  • Epstein-Barr virus (EBV) serology (VCA IgG)

    EBV serology will be assessed (VCA IgG) in the serum of patients to evaluate the variation of antibody titers over time (at baseline, at 6- and 12-month follow-up), and compare to titers at the time of diagnosis (when available in their medical record).

    12 months after baseline

Study Arms (2)

PIRA

EXPERIMENTAL

From the MS functional outcome database, identification of a cohort of patients with RMS experiencing progression independent of relapse (PIRA)

Device: Visual Evoked Potential (VEP)Device: Somatosensory evoked potential (SSEP)Device: Transcranial magnetic motor evoked potentials (TCmMEP)Device: Tesla Brain MRIDiagnostic Test: Blood test - Neurofilament light chain (NfL)Diagnostic Test: Blood test - EBV serology

N-PIRA

ACTIVE COMPARATOR

From the MS functional outcome database, identification of a cohort of patients with RMS not experiencing progression independent of relapse (N-PIRA)

Device: Visual Evoked Potential (VEP)Device: Somatosensory evoked potential (SSEP)Device: Transcranial magnetic motor evoked potentials (TCmMEP)Device: Tesla Brain MRIDiagnostic Test: Blood test - Neurofilament light chain (NfL)Diagnostic Test: Blood test - EBV serology

Interventions

To assess the integrity of visual pathways through the optic nerves to the visual cortex, latencies and amplitudes of P100 will be measured after pattern-reversal stimuli.

N-PIRAPIRA

To assess the integrity of sensitive pathways through the peripheral nerves and dorsal spinal cord to the somatosensory cortex. For the upper limbs, latencies and amplitudes of N9, N13, P14, N20 and P25 will be measured after median nerve stimulation. For the lower limbs, latency and amplitude of P40 will be measured after tibial nerve stimulation.

N-PIRAPIRA

To measure the integrity of motor pathways, the central conduction times will be measured for upper and lower limbs through magnetic stimulations of the primary motor cortex and the spinal cord, at cervical and lumbar levels.

N-PIRAPIRA

All patients will undergo a single brain MRI on a 3T scanner. The acquisition protocol will include high-resolution three-dimensional (3D) T2\*-weighted echo-planar imaging and 3D T2-FLAIR images acquired, respectively, during or after intravenous injection of a single dose (0.1mmol/kg) of gadolinium-based contrast material.

N-PIRAPIRA

\- Neurofilament light chain (NfL) will be tested (Quanterix's Simoa® Technology) in the serum of patients. To evaluate their variation over time, 3 time-point tests will be obtained at 6 months apart (at baseline, at 6- and 12-month follow-up).

N-PIRAPIRA

EBV serology will be assessed (VCA IgG) in the serum of patients to evaluate the variation of antibody titers over time (at baseline, at 6- and 12-month follow-up), and compare to titers at the time of diagnosis (when available in their medical record).

N-PIRAPIRA

Eligibility Criteria

Age18 Years+
Sexall
Healthy VolunteersNo
Age GroupsAdult (18-64), Older Adult (65+)

You may qualify if:

  • Patients ≥ 18-year-old with diagnosis of RMS according to 2017 McDonald diagnostic criteria
  • Availability in the functional outcome database of at least 3 time-point complete evaluations with a time frame from the first to the last evaluation of minimum 12 months
  • Most recent functional outcome evaluation within 12 months of enrollment
  • Availability of follow-up MRI data during the observational period

You may not qualify if:

  • a) Contraindication to one or more of the paraclinical tests of the prospective multimodal evaluation

Contact the study team to confirm eligibility.

Sponsors & Collaborators

Study Sites (1)

CHU Brugmann

Brussels, 1020, Belgium

RECRUITING

MeSH Terms

Conditions

Multiple Sclerosis

Condition Hierarchy (Ancestors)

Demyelinating Autoimmune Diseases, CNSAutoimmune Diseases of the Nervous SystemNervous System DiseasesDemyelinating DiseasesAutoimmune DiseasesImmune System Diseases

Central Study Contacts

Study Design

Study Type
interventional
Phase
not applicable
Allocation
NON RANDOMIZED
Masking
NONE
Purpose
DIAGNOSTIC
Intervention Model
PARALLEL
Sponsor Type
OTHER
Responsible Party
PRINCIPAL INVESTIGATOR
PI Title
Head of neurology department

Study Record Dates

First Submitted

May 15, 2023

First Posted

July 12, 2023

Study Start

February 14, 2023

Primary Completion

December 31, 2025

Study Completion

December 31, 2025

Last Updated

July 12, 2023

Record last verified: 2023-07

Data Sharing

IPD Sharing
Will not share

Locations