NCT05353517

Brief Summary

The pathophysiological mechanisms underlying Movement Disorders, including Parkinson's disease, have been related to altered synaptic plasticity affecting several structures of the central nervous system. Although several previous neurophysiologic investigations have shown abnormal long-term potentiation and depression-like plasticity in M1, other regions crucially involved in motor planning and execution, including the spinal cord, have been studied less. Parkinson's disease arises from the progressive loss of dendritic spines followed by atrophy of specific cortical (i.e. M1) and subcortical structures (i.e. putamen). These structural changes are responsible for the main clinical features of PD such as bradykinesia and rigidity. The present research project aims to probe non-invasively the main pathophysiologic mechanisms underlying altered synaptic plasticity in M1 and spinal cord and their relationship in a cohort of patients with movement disorders, including Parkinson's disease. More in detail, the investigators will use specific methodologies able to induce plasticity, including the repetitive transcranial magnetic stimulation (TMS), concerning the M1 and the focal muscle vibration, regarding the spinal cord. The neuromodulation protocol will imply 2 separate sessions, randomly scheduled to take into account the effect of the symptomatic pharmacologic treatment. Furthermore, patients will be randomly assigned to sham or real non-invasive stimulation groups. Before and after the stimulation protocol, the investigators will collect specific clinical as well as neurophysiologic measures (i.e., thresholds) according to standardized procedures. In conclusion, the goal of the study is to investigate the abnormal plasticity in the M1 and spinal cord in patients affected by specific movement disorders, through non-invasive techniques.

Trial Health

43
At Risk

Trial Health Score

Automated assessment based on enrollment pace, timeline, and geographic reach

Trial has exceeded expected completion date
Enrollment
50

participants targeted

Target at P25-P50 for all trials

Timeline
Completed

Started Dec 2022

Geographic Reach
1 country

1 active site

Status
unknown

Health score is calculated from publicly available data and should be used for screening purposes only.

Trial Relationships

Click on a node to explore related trials.

Study Timeline

Key milestones and dates

First Submitted

Initial submission to the registry

April 20, 2022

Completed
9 days until next milestone

First Posted

Study publicly available on registry

April 29, 2022

Completed
7 months until next milestone

Study Start

First participant enrolled

December 1, 2022

Completed
1 month until next milestone

Primary Completion

Last participant's last visit for primary outcome

December 31, 2022

Completed
1 year until next milestone

Study Completion

Last participant's last visit for all outcomes

December 31, 2023

Completed
Last Updated

March 23, 2023

Status Verified

March 1, 2023

Enrollment Period

1 month

First QC Date

April 20, 2022

Last Update Submit

March 21, 2023

Conditions

Outcome Measures

Primary Outcomes (5)

  • Analysis of neurophysiologic measures

    Rest motor threshold

    baseline

  • Analysis of neurophysiologic measures

    Active motor threshold

    baseline

  • Analysis of neurophysiologic measures

    Amplitude of motor evoked potentials

    through study completion, an average of 1 year

  • Analysis of neurophysiologic measures

    H reflex

    through study completion, an average of 1 year

  • Analysis of neurophysiologic measures

    input-output curve

    through study completion, an average of 1 year

Study Arms (2)

Healthy Subjects

Device: focal muscle vibration to induce spinal plasticity theta burst stimulation and paired associative stimulation to induce plasticity in M1

Device: Device: focal muscle vibration and theta burst stimulation

Patients with Parkinson's disease

Device: theta burst stimulation and paired associative stimulation to induce plasticity in M1 Device: focal muscle vibration to induce spinal plasticity

Device: Device: focal muscle vibration and theta burst stimulation

Interventions

non invasive techniques of neuromodulation for inducing plasticity in M1 and spinal cord

Healthy SubjectsPatients with Parkinson's disease

Eligibility Criteria

Age18 Years+
Sexall
Healthy VolunteersYes
Age GroupsAdult (18-64), Older Adult (65+)
Sampling MethodProbability Sample
Study Population

25 patients with PD 25 HS

You may qualify if:

  • clinical diagnosis of Parkinson's disease

You may not qualify if:

  • psychiatric disorders, including severe congnitive loss and depression

Contact the study team to confirm eligibility.

Sponsors & Collaborators

Study Sites (1)

IRCCS Neuromed

Pozzilli, 86077, Italy

RECRUITING

MeSH Terms

Conditions

Movement DisordersParkinson Disease

Condition Hierarchy (Ancestors)

Central Nervous System DiseasesNervous System DiseasesParkinsonian DisordersBasal Ganglia DiseasesBrain DiseasesSynucleinopathiesNeurodegenerative Diseases

Central Study Contacts

Antonio Suppa, MD, PhD

CONTACT

Francesco Asci, MD

CONTACT

Study Design

Study Type
observational
Observational Model
CASE CONTROL
Time Perspective
CROSS SECTIONAL
Sponsor Type
OTHER
Responsible Party
PRINCIPAL INVESTIGATOR
PI Title
Prof, MD, PhD

Study Record Dates

First Submitted

April 20, 2022

First Posted

April 29, 2022

Study Start

December 1, 2022

Primary Completion

December 31, 2022

Study Completion

December 31, 2023

Last Updated

March 23, 2023

Record last verified: 2023-03

Locations